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Dassault Systemes discovery studio visualizer version 2016
Discovery Studio Visualizer Version 2016, supplied by Dassault Systemes, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/discovery+studio+visualizer+version+2016/discovery+studio+modeling+environment+release+2017/pm40371931-665-12-17
Average 90 stars, based on 1 article reviews
discovery studio visualizer version 2016 - by Bioz Stars, 2026-11
90/100 stars

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Related Articles

Clinical Proteomics:

Article Title: Design, Synthesis, and Biological Evaluation of 4-(Difluoromethyl)-1 H -imidazole-5-carboxylic Acids/4-(3-Cyanophenoxy)pyrimidine-5-carboxylic Acids as P2Y 1 Receptor Antagonists for Ischemic Stroke Treatment.
Article Snippet: To develop effective agents for ischemic stroke, compounds 19 (IC50 = 0.49 μM) and 36b (IC50 = 0.50 μM), as analogues of our previously reported antiplatelet agent HNW001, were identified as potent P2Y1 receptor antagonists, which were superior to HNW001 and BPTU (IC50 = 4.07 and 2.50 μM, respectively).. Notably, these two compounds showed remarkable neuroprotective potency against oxidative stress by upregulating nuclear Nrf2 protein levels in vitro.. Additionally, compounds 19 and 36b demonstrated favorable blood-brain barrier penetration potential in rats, and the rat MCAO model showed that they could effectively reduce infarction sizes with ED50 values of 8.39 and 4.60 mg/kg, respectively.

Protein Binding:

Article Title: Design, Synthesis, and Biological Evaluation of 4-(Difluoromethyl)-1 H -imidazole-5-carboxylic Acids/4-(3-Cyanophenoxy)pyrimidine-5-carboxylic Acids as P2Y 1 Receptor Antagonists for Ischemic Stroke Treatment.
Article Snippet: To develop effective agents for ischemic stroke, compounds 19 (IC50 = 0.49 μM) and 36b (IC50 = 0.50 μM), as analogues of our previously reported antiplatelet agent HNW001, were identified as potent P2Y1 receptor antagonists, which were superior to HNW001 and BPTU (IC50 = 4.07 and 2.50 μM, respectively).. Notably, these two compounds showed remarkable neuroprotective potency against oxidative stress by upregulating nuclear Nrf2 protein levels in vitro.. Additionally, compounds 19 and 36b demonstrated favorable blood-brain barrier penetration potential in rats, and the rat MCAO model showed that they could effectively reduce infarction sizes with ED50 values of 8.39 and 4.60 mg/kg, respectively.



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